Alzheimers increased with  Early Retirement

 

This research came from a french study. Alzheimers is a degenerative disease of the brain and parkinsons, huntingtons chorea and ALS are other degenerative disease. Primarily these are caused by damage to the brain with toxins in our food. These toxins come from Animal Foods, Toxic metals (Mercury, lead and cadmium) food additives such as MSG and Aspartame.  Yet the brain is protected by gainful employment.  Brain needs exercises just like muscles. Working provides the exercises for the brain. This is especially true if you like the work you do. We live in a society and the most satisfying work is when your work contributes to happiness of other people.

BOSTON -- French retirees who had stopped working relatively late in life were less likely to develop Alzheimer's disease, a researcher reported here.

Analysis of a French healthcare insurer's records indicated that, for each year after age 60 at which a person retired, the risk of subsequently developing Alzheimer's disease was lower by 3.2% (HR 0.968, 95% CI 0.962-0.973), said Carole Dufouil, PhD, of INSERM in Bordeaux, France.

After adjusting for certain other risk factors, individuals retiring at 65 were 14.6% less likely to develop Alzheimer's disease than those retiring at 60, she said at a press briefing held prior to her formal presentation at the Alzheimer's Association International Conference.

The results were "in line with the use-it-or-lose-it hypothesis," she said, which holds that people who remain mentally active develop dementia at lower rates than those who don't.

Data for the study came from an insurance provider for self-employed workers in France, mainly shopkeepers and craft workers, Dufouil explained. Records for some 430,000 pensioners as of December 2010 were analyzed, including 11,397 who were considered to have developed Alzheimer's disease or related dementias after retirement.

The analysis excluded individuals whose records indicated a Parkinson's disease diagnosis at any time and also those with apparent dementia at retirement.

Onset of Alzheimer's disease was defined as a diagnostic code of ALD15 in the French healthcare system, which refers to "Alzheimer's disease and related disorders," or purchase of anti-dementia drugs such as memantine (Namenda) or acetylcholinesterase inhibitors.

Because they had such a large data set to work with, Dufouil and colleagues also conducted analyses to determine if the relationship between retirement age and Alzheimer's risk differed among subgroups.

There were statistically significant differences in two groups -- men versus women and craft workers versus shopkeepers -- but all still showed a significant decrease in risk of at least 2% for each year beyond age 60 for retirement.

Stratification by age of birth or age at Alzheimer's disease diagnosis also did not make a great difference in results, Dufouil said.

She added that sensitivity analyses in which some individuals were excluded -- such as those with diagnoses within 5 or 10 years of retirement, those retiring after age 75, and those with relatively short periods of self-employment before retirement -- also confirmed the topline results.

Dufouil noted several limitations to the study: The findings might not be applicable to other occupational types, the definition of new-onset dementia could be questioned, and the data lacked information on formal educational attainment and certain other important risk factors, she said.

Nevertheless, she said the results added to the growing body of evidence that "maintaining high levels of cognitive and social stimulation" is beneficial in seniors.

Emerging research on the widespread degenerative brain disease known as Alzheimer’s suggests that this prevalent form of dementia is actually a type of diabetes. Published in the Journal of Alzheimer’s Disease, a recent study out of Rhode Island Hospital (RIH) confirms that Alzheimer’s is marked by brain insulin resistance and corresponding inflammation, a condition that some researchers are now referring to as type-3 diabetes

Te human body needs cholesterol in order to stay healthy. If your cholesterol levels are too high, or if cholesterol is clogging your arteries, it means that your body has an inflammation problem that is preventing the proper absorption and use of cholesterol. Cholesterol is not the problem, in other words — your body’s chronic inflammation is the problem.

Consuming more healthy saturated fats like coconut oil can not only help repair the inflammation problem that promotes the progression of Alzheimer’s, but it can also increase the absorption of cholesterol in the brain, which in turn promotes healthy neuronal function. (http://www.naturalnews.com) Such advice runs contrary to the mainstream medical system’s misguided philosophies about health, but science actually shows that the human body requires saturated fats and cholesterol, and that these fats are vital for maintaining optimal brain health.