What causes cancer and how to prevent it.

The War on Cancer

War on cancer has dragged on far too long and at too great a human cost.
The internationally organized Human Genome Project was a huge success, and in 2006 the United States went on to fund The Cancer Genome Atlas with the hope of zeroing in on a cure for cancer.
By 2015, after $375 million had been spent, the DNA (deoxyribonucleic acid, a cell’s genetic instructions) of 10,000 tumors had been sequenced, and 10 million cancer-related mutations had been identified.
Most of the mutations formed a bewildering hodgepodge of genetic oddities, with little commonality between tumors. Cancers are often quick to become resistant, typically by activating different genes to bypass whatever cellular process is blocked by the treatment.


Cancer cells preferentially ferment glucose in the cytoplasm even when there is sufficient oxygen to generate energy using the more efficient mitochondrial pathway. In this scenario, it’s easy to understand that cancer is a disease of mitochondrial metabolism.
Warburg’s theory was based on his findings that all cancer cells have some defect in their ability to use oxygen to obtain energy through mitochondrial respiration.



As the tumor grows, it restricts the flow of blood containing oxygen and other vital nutrients. A cancer cell’s ability to ferment glucose allows it to survive and thrive in a hypoxic (low oxygen) environment.
Rate of glycolysis in cancer cells is typically 10 to 15 times the rate in a normal cell. For that to occur, cancer cells need a way to allow more transport of glucose into the cell.
Cytoplasmic hybrid cells, also known as cybrids, were created by transferring the nuclei of tumor cells into cells with normal cytoplasm. The result: cells that replicated and survived, despite having nuclei with mutated DNA. Other cybrids were created by transferring normal nuclei into tumor cells containing cytoplasm that displayed the Warburg effect (excessive fermentation of glucose). These cells also replicated, but the majority of them did not survive. The information presented here supports the notion that cancer originates from damage to the mitochondria in the cytoplasm rather than from damage to the genome in the nucleus.
Immune surveillance is the task of seeking out and destroying our own dysfunctional or damaged cells.



All cancer takes advantage of mutations that allow it to evade destruction by the immune system.
Radiation and many chemotherapy drugs are prooxidant therapies: that is, they work by overwhelming dysfunctional cancer cells, creating so much ROS that the therapy directly kills cancer cells. But there’s collateral damage to neighboring healthy cells as well, and this collective damage contributes to widespread inflammation.



Chemotherapy, for example, targets cells that are replicating quickly (cancer cells), though unfortunately it also causes the death of the healthy fast-growing cells that line your digestive tract from “stem to stern” (mouth to anus). In turn, this disrupts your microbiome, which creates problems with appetite, digestion, and the absorption of nutrients. The conventional nutrition support offered by your oncology team is designed primarily to remediate these gastrointestinal side effects. When you consider that at least 70 percent of your body’s immune defense arises from activity within the gut, you begin to get a clearer picture of the domino effect associated with conventional therapies.

Chemotherapy and radiation cause inflammation to skyrocket. That’s why so many treatment protocols include pre- and post-treatment inflammation-suppressing steroid medications. Unfortunately, these medications also suppress immune function, a very undesirable side effect that lowers the body’s resistance to everyday infections. Steroids also raise blood glucose levels, another detrimental side effect. Furthermore, some protocols also call for the use of prophylactic antibiotics. And, guess what? Antibiotics have side effects as well, including total disruption of your protective gut microbiome.


Insulin Resistance Adds Fuel to the Flames
Elevated levels of blood glucose and insulin are the markers used to diagnose diseases of insulin resistance, including prediabetes, type 2 diabetes, and polycystic ovary syndrome (PCOS). These markers are also associated with a higher risk of many cancers.
Insulin-resistant fat cells secrete inflammatory cytokines (including those involved in the immune response). This results in a chronic inflammatory state that damages cellular membranes and DNA while simultaneously altering normal mitochondrial activities, including molecular signaling and repair mechanisms, thereby opening more avenues for cancer progression.
Medicine is still heavily reliant on traditional interventions designed to poison cancer. In his book Tripping over the Truth, science writer Travis Christofferson describes the work of doctors who were researching the therapeutic potential of “war gas” (nitrogen mustard).
Most important lesson for us to learn is that most cancers start because of the wrong diet. Similarly heart disease, strokes and other chronic diseases start due to the same reason.


Today the foods we eat lack the antioxidants and the vitamins they used to have centuries ago. So we need extra help to prevent chronic diseases. Please call 0772300454 for more information. If you already have diabetes or hypertension and wants to find out how to cure it naturally there is help available to you.